This talk enumerates the challenges regarding data accessibility and reusability inherent in the current scientific publication system, and discusses novel approaches to these challenges, such as the EBRAINS Live Papers platform. 

This brief video gives an introduction to the eighth session of INCF's Neuroinformatics Assembly 2023, focusing on FAIR data and the role of academic journals. 

This talk gives an overview of the perspectives and FAIR-aligned policies of the academic journal Public Library of Science, better known as PLOS. This journal is a nonprofit, open access publisher empowering researchers to accelerate progress in science. 

This talk highlights a set of platform technologies, software, and data collections that close and shorten the feedback cycle in research. 

This lecture covers visualizing extracellular neurotransmitter dynamics

This lesson goes over the basic mechanisms of neural synapses, the space between neurons where signals may be transmitted. 

This lesson describes spike timing-dependent plasticity (STDP), a biological process that adjusts the strength of connections between neurons in the brain, and how one can implement or mimic this process in a computational model. You will also find links for practical exercises at the bottom of this page. 

This lesson discusses a gripping neuroscientific question: why have neurons developed the discrete action potential, or spike, as a principle method of communication? 

This lecture consists of the second half of the introduction to signal transduction, here focusing on cell receptors and signalling cascades.

In this lesson, you will learn about GABAergic interneurons and local inhibition on the circuit level.

This lecture focuses on how the immune system can target and attack the nervous system to produce autoimmune responses that may result in diseases such as multiple sclerosis, neuromyelitis, and lupus cerebritis manifested by motor, sensory, and cognitive impairments. Despite the fact that the brain is an immune-privileged site, autoreactive lymphocytes producing proinflammatory cytokines can cause active brain inflammation, leading to myelin and axonal loss.